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#11
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![]() Arguing with Levac is like arguing with Fred Hahn. I don't know which one is worse.
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James Krieger Weightology, LLC Empowering you with knowledge for weight loss success The Health Sleuth Journal of Pure Power |
#12
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![]() Why argue then, its like discussing with a pastor from some obscure religious sect - You'll never ever convince them, no matter of what solid scientific evidence you put on their table...
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#13
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![]() At least it's nice to have someone proving that they were wrong
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#14
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#15
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Here's a study comparing a low-fat, high carbohydrate diet to a carbohydrate-free, high fat diet in rodents. The net increase in epididymal fat was identical in both conditions, which refutes Martin's claim that one cannot gain fat on a carbohydrate-free diet. The researchers stated: In summary, we have demonstrated that triglyceride synthesis in adipose tissue is not down-regulated when carbohydrate appears to be limiting; there is an extensive triglyceride turnover during conditions where ~0% of the calories are derived from dietary carbohydrate. Our data suggest that the contribution of glucose and non-glucose carbon sources shifts with changes in nutritional status
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James Krieger Weightology, LLC Empowering you with knowledge for weight loss success The Health Sleuth Journal of Pure Power |
#16
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I wish it wasn't the case, but I just think the ONLY point of arguing with the crazy crowds is to win over the swing crowds who watch the arguments. |
#17
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James Krieger Weightology, LLC Empowering you with knowledge for weight loss success The Health Sleuth Journal of Pure Power |
#18
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![]() I think it's disingenuous to try to have it both ways. If you're not going to accept rat work in support of something, don't accept it to dismiss something. Rat work is simply irrelevant in this case.
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#19
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However, I don't think it's disingenuous to accept rat models under certain circumstances and be extremely cautious with or dismiss them under other circumstances. It depends upon what is being examined, and the magnitude of the differences between rat and human physiology and whether those differences are relevant to the problem at hand. For example, rats have a greater capacity for de novo lipogenesis than humans, which is why some of the high fructose feeding rodent studies are not completely relevant to humans (particularly given that the fructose contents of the diets are often ridiculously high). However, in this particular case, I do think the rat model is relevant. It's a model examining the contribution of glucose and non-glucose sources of glycerol for triglyceride synthesis under different dietary conditions. I don't think the differences in rat and human physiology would have a great impact on the applicability of these results to humans. The low carb zealots argue that carbohydrate is required for fat synthesis as a glycerol source. This paper makes it clear that adipose tissue "finds a way". In the presence of high carbohydrate, it will synthesize glycerol from glucose. But in the presence of high fat and no carbohydrate, it will synthesize glycerol through glyceroneogenesis from the ingested triglycerides. And one pathway is not more efficient than the other. I think it comes down to why someone is dismissing or accepting a rat model.
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James Krieger Weightology, LLC Empowering you with knowledge for weight loss success The Health Sleuth Journal of Pure Power |
#20
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![]() I just saw this thread, very nice to have someone else arguing with Low Carb fruitcakes. I don't understand, why do those people defend their views so much? It's like they're on some super "high" by following their low and zero carb diets?
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